Tumor Aneuploidy (häftad)
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Format
Häftad (Paperback / softback)
Språk
Engelska
Antal sidor
141
Utgivningsdatum
1985-11-01
Förlag
Springer-Verlag Berlin and Heidelberg GmbH & Co. K
Medarbetare
Bloomfield, Clara (ass. by)/Andreeff, Michael (ass. by)/Barlogie, B. (ass. by)/Bassewitz, D.B. von (ass. by)/Becher, R. (ass. by)/Bloomfield, Clara (ass. by)/Andreeff, Michael (ass. by)/Barlogie, B. (ass. by)/Bassewitz, D.B. von (ass. by)/Becher, R. (ass. by)/Bloomfield, Clara (ass. by)/Andreeff, Mi
Illustrationer
VIII, 141 p.
Dimensioner
244 x 170 x 8 mm
Vikt
254 g
Antal komponenter
1
Komponenter
1 Paperback / softback
ISBN
9783540153764

Tumor Aneuploidy

av Thomas Buchner, Clara D Bloomfield, Wolfgang Hiddemann, Dieter K Hossfeld, Johannes Schumann
Häftad,  Engelska, 1985-11-01
783
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Chromosome abnormalities of cancer cells have been recognized for a long time, and have generally proven to be a highly specific marker ofmalignancy. The contri- butions collected in this book, "Tumor Aneuploidy", cover several major aspects of present knowledge conceming the occurrence and clinical significance of chromo- some abnormalities as delineated by karyotype analyses or measurements of the cellular DNA content. Certain non-random clonal chromosome losses, deletions and translocations ap- pear to represent primary genetic lesions of malignancies and reflect their clonal origin. Secondary intraneoplastic genetic evolution is suggested by major clonal ab- normalities of chromosome number and cellular DNA content. Both types of ge- netic changes have been reaching great relevance in cancer medicine, today. Although the Philadelphia chromosome was first discovered in chronic myelo- cytic leukemia (CML), by Nowell and Hungerford in 1960, new banding techniques developed in the 1970's were needed to identity this abnormality as a translocation between chromosomes 9 and 22 (t(9; 22)). Soon thereafter, further non-random translocations were detected and attributed to special diseases like t(8; 21) and t(15; 17) to acute myeloid leukemias (AML) and t(9; 22), t(4; 11), t(8; 14) to acute lymphoblastic leukemia (ALL).
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Thomas Buchner, Clara D Bloomfield, Wolfgang Hiddemann, Dieter K Hossfeld, Johannes Schumann

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