Jacques Philippon – författare

Joseph Babinski''s contributions to French medicine have been well-documented, but there has yet to be a significant and an authoritative biography of him--until now. Two French physicians, Jaques Philippon and Jacques Poirier, analyze Babinski''s great scientific achievements, explore his unique family history, and publish, for the first time, a complete bibliography of his publications. The "Babinski sign," considered as his greatest diagnostic achievement, is typically one of the first neurological tests performed by a specialist or primary care physician to determine the existence of an injury to the pyramidal tract. Joseph Babinski, however, is more than just the "Babinski sign" that has made him famous and revered. As the authors explain, he was an early contributor to the fields of cutaneous and tendinous reflexes, cerebellar and vestibular semiology, hysteria and pithitiasm, localization of spinal cord compressions, and the birth of French neurosurgery. This book chronicles his family''s emigration from Poland to France, his tutelage and early career under great teachers such as Alfred Vulpian, Victor Cornil, and Jean-Martin Charcot at the Hopital de la Salpetriere in Paris, his methods and observations during 27 years as department head at La Pitie, as well as the close and unique relationship with his brother, Henri, the well-known Ali-Bab. Finally, Babinski''s life and times can be accessed in one fresh and intriguing book!

Joseph Babinski''s contributions to French medicine have been well-documented, but there has yet to be a significant and an authoritative biography of him--until now. Two French physicians, Jaques Philippon and Jacques Poirier, analyze Babinski''s great scientific achievements, explore his unique family history, and publish, for the first time, a complete bibliography of his publications. The "Babinski sign," considered as his greatest diagnostic achievement, is typically one of the first neurological tests performed by a specialist or primary care physician to determine the existence of an injury to the pyramidal tract. Joseph Babinski, however, is more than just the "Babinski sign" that has made him famous and revered. As the authors explain, he was an early contributor to the fields of cutaneous and tendinous reflexes, cerebellar and vestibular semiology, hysteria and pithitiasm, localization of spinal cord compressions, and the birth of French neurosurgery. This book chronicles his family''s emigration from Poland to France, his tutelage and early career under great teachers such as Alfred Vulpian, Victor Cornil, and Jean-Martin Charcot at the Hopital de la Salpetriere in Paris, his methods and observations during 27 years as department head at La Pitie, as well as the close and unique relationship with his brother, Henri, the well-known Ali-Bab. Finally, Babinski''s life and times can be accessed in one fresh and intriguing book!

During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by its title "Prevention and treatment of delayed ischaemic dysfunction in patients with subarachnoid haemorrhage: an update", papers presented at the Symposium covered a broad spectrum from some basic data on pathophysiology of subarachnoid haemorrhage (SAH) and delayed ischaemic dysfunction, to the clinical use of nimodipine, which has been largely documented among calcium inhibitors for its cerebrovascular properties. This supplementum of Acta Neurochirurgica is based upon the papers presented. Some articles, however, have been extended to take into account further results in order to present a broad view of the subject. The Symposium started with two presentations concerning general aspects of SAH: in the first one, B. Voldby reviewed the pathophysiological events following SAH: if modifications of intracranial pressure (ICP) and decrease of cerebral blood flow (CBF) are the primary alterations, metabolic changes (particularly reduction in CMR0 ) 2 contribute rapidly to disturbances of brain circulation. A variety of mechanisms may be responsible for the neurological dysfunction: most of them are directly related to the reduction in cerebral perfusion pressure (CPP) which may result in ischaemia. This fall in CPP (either due to elevation of ICP or reduction of mean arterial blood pressure) is observed under different pathophysiological conditions, discussed in the paper by A. D. Mendelow.