hypothesis that proposes that defects in PHD2 and PHD3 function may be causal of the symptoms of schizopnrenia.

AvJohn Neville

Häftad, Engelska, 2019

83 kr

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Produktinformation

Utgivningsdatum:2019-09-08
Mått:108 x 178 x 2 mm
Vikt:36 g
Format:Häftad
Språk:Engelska
Antal sidor:42
Förlag:Lulu.com
ISBN:9780244516628

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Mir190, perhaps via interactions involving TP53INP1, CBX4 and 14-3-3 proteins, may play a significant role in SEC24A/SEC24D and CSNK1A/CSNK1D COP2 interactions which may be of significance in the aetiology of schizophrenia.

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CCNB1, CCNB2, CCNA1, CCNA2, SYT1, SYT2, CKS2, CKS1B, CCNB3, SKP1, CDK1, RPS23, RPS27A, ZFAND4, RPS27, RPS27l, BUB1, BUB1B could play significant roles in the aetiology of schizophrenia by acting as points of contact between ALDH18A1 and SEC23IP (COP2).

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majority of 219 genes predicted to be HIF1 targets have altered expression/function in schizopnrenia, or are associated with schizopnrenia, meaning that altered/defective HIF1 targeting must play a central role in the pathogenesis of schizophrenia

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