Häftad, Engelska, 2018
Biophysical Basis of Physiology and Calcium Signaling Mechanism in Cardiac and Smooth Muscle
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Beskrivning
Biophysical Basis of Physiology and Calcium Signaling Mechanism in Cardiac and Smooth Muscle acts as a bridge between physiology and physics by discussing the physiology and calcium signaling mechanism in cardiac and smooth muscle. By exploring the mechanism of the cyclic release of stored Ca^(2+) in the SR or ER, this book covers the cell communication system, including excitable cells, recognizing the most relevant mechanisms of cell communication. Serving as a bridge between physiology and physics, coverage spans the physiology and calcium signaling mechanism in cardiac and smooth muscle, offering insight to physiological scientists, pharmaceutical scientists, medical doctors, biologists and physicists.
- Explores the mechanism of the cyclic release of stored Ca^2+ in the SR or ER
- Provides in-depth coverage of cell communication systems to explain the most relevant mechanisms of cell communication
- Covers the physiology and calcium signaling mechanism in cardiac and smooth muscle
Produktinformation
- Utgivningsdatum: 2018-02-15
- Mått: 191 x 235 x undefined mm
- Vikt: 480 g
- Format: Häftad
- Språk: Engelska
- Antal sidor: 232
- Förlag: Elsevier Science
- ISBN: 9780128149508
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Tetsuya Watanabe is President of the Watanabe Institute of Mathematical Pathophysiology in Hamamatsu, Japan. His professional background includes postdoctoral fellowship and faculty appointments in the Department of Pharmacology at the University of Pennsylvania School of Medicine in Philadelphia. His work focuses on mathematical pathophysiology, physiology, electrochemical signaling, excitable cells, and stress- and lifestyle-associated diseases. He is the author of books published by Academic Press/Elsevier, including works on calcium signaling in cardiac and smooth muscle, electrochemical dynamics in excitable cells, and the clinical pathophysiology of hypertension, diabetes, and other lifestyle-associated diseases.
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