Two groups of volunteers having no eye disease were examined electro-oculo- graphically twice at ten minute intervals. One group smoked in the break, one didn't. In the non-smoking group there are a number of significant correlations between the tested parameters in the dark-period and in the light-period, which could not be found to the same degree in the smoking group. The fact that there were differences between the two groups-even though not interpretable systematicaIly-suggests, that smoking influences physiological processes in the EOG. In the second period of the test, a parallelism in the course of EOG and EEG was observed, the EEG-changes, however, are interpreted more psychologically than pharmacologicaIly. REFERENCES HAASE, E. & MUELLER, W. Messbare Beeinflussung des EOG durch Zigarettenrauchen1 Klin. Mbl. Augenhk. Bd. 158:677 (1971). HAGER, H. Thrombangiitis obliterans und Auge. Klin. Mbl. Augenhk. Bd. 114:238 (1949). HAUSER, H., SCHWARZE, B. E., ROTH, G. & BICKFORD, R. G. Electroencephalographic changes related to smoking. EEG clin. Neurophysiol. Vol. 10:576P (1958). HEIMBOCK. Wien. klin. Wschr. Bd. 73:529 (1961). HOLLWICH, F., JUNEMANN, G. & DAMASKE, E. Auge.In: Nikotin. Edited by Schievelbein. H. Georg Thieme Verlag, Stuttgart (1968). HOMER, L. D. & KOLDER, H. Mathematical model of oscillations in human corneo-retinal potential. Pflugers Arch. ges. Physiol. 287 :197 (1966). KOLDER H. Spontane und experimentelle Anderungen des Bestandpotentials des menschlichen Auges. P{lugerrs Arch. ges. Physiol. 268:258 (1959). KOLDER, H. E. & HOCHGESAND, P. Empirical Model of Electro-Oculogram. Doc. Ophtha!. (1972) in press.
